Inspiratory muscle training abolishes the blood lactate increase associated with volitional hyperpnoea superimposed on exercise and accelerates lactate and oxygen uptake kinetics at the onset of exercise

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Brown, P.I., Sharpe, G.R. ORCID: 0000-0002-4575-2332 and Johnson, M.A. ORCID: 0000-0002-8226-9438, 2012. Inspiratory muscle training abolishes the blood lactate increase associated with volitional hyperpnoea superimposed on exercise and accelerates lactate and oxygen uptake kinetics at the onset of exercise. European Journal of Applied Physiology, 112 (6), pp. 2117-2129. ISSN 1439-6319

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Official URL: http://dx.doi.org/10.1007/s00421-011-2185-8

Abstract

We examined the effects of inspiratory muscle training (IMT) upon volitional hyperpnoea-mediated increases in blood lactate ([lac−]B) during cycling at maximal lactate steady state (MLSS) power, and blood lactate and oxygen uptake kinetics at the onset of exercise. Twenty males formed either an IMT (n=10) or control group (n=10). Before and after a 6-wk intervention two 30 min trials were performed at MLSS (207 ± 28 W), which was determined using repeated 30 min constant power trials. The first was a reference trial, whereas during the second trial from 20-28 min participants mimicked the breathing pattern commensurate with 90% of the maximal minute ventilation (V ˙ E) measured during maximal incremental exercise. Before the intervention the MLSS [lac−]B was 3.7 ± 1.8 and 3.9 ± 1.6 mmol·L-1 in the IMT and control group, respectively.

Item Type:

Journal article

Publication Title:

European Journal of Applied Physiology

Creators:

Brown, P.I., Sharpe, G.R. and Johnson, M.A.

Publisher:

Springer-Verlag

Place of Publication:

Berlin

Date:

2012