Items where Author is "Hyland, P"

DEBOWSKA, A., BODUSZEK, D., HYLAND, P. and GOODSON, S., 2014. Biological correlates of psychopathy: a brief review. Mental Health Review Journal, 19 (2), pp. 110-123. ISSN 1361-9322

MARTHANDAN, S., HYLAND, P., PAWELEC, G. and BARNETT, Y., 2013. An investigation of the effects of the antioxidants, ebselen or N-acetyl cysteine on human peripheral blood mononuclear cells and T cells. Immunology and Ageing, 10. ISSN 1742-4933

KUNZMANN, A., LIU, D., ANNETT, K., MALAISÉ, M., THAA, B., HYLAND, P., BARNETT, Y.A. and BURKLE, A., 2006. Flow-cytometric assessment of cellular poly(ADP-ribosyl)ation capacity in peripheral blood lymphocytes. Immunity and Ageing, 3: 8.

ANNETT, K.E., DUGGAN, O., FREEBURN, R., HYLAND, P., PAWELEC, G. and BARNETT, Y.A., 2005. An investigation of DNA mismatch repair capacity under normal culture conditions and under conditions of supra-physiological challenge in human CD4+T cell clones from donors of different ages. Experimental Gerontology, 40 (12), pp. 976-981. ISSN 0531-5565

DUGGAN, O., HYLAND, P., ANNETT, K., FREEBURN, R., BARNETT, C., PAWELEC, G. and BARNETT, Y., 2004. Effects of a reduced oxygen tension culture system on human T cell clones as a function of in vitro age. Experimental Gerontology, 39, pp. 525-530.

ANNETT, K., HYLAND, P., DUGGAN, O., BARNETT, C. and BARNETT, Y., 2004. An investigation of DNA excision repair capacity in human CD4+ T cell clones as a function of age in vitro. Experimental Gerontology, 39, pp. 491-498.

ROSS, O.A., CURRAN, M.D., MIDDLETON, D., MCILHATTON, B.P., HYLAND, P., DUGGAN, O., ANNETT, K., BARNETT, C. and BARNETT, Y., 2003. Genetic damage and ageing T cells. Advances in Cell Aging and Gerontology, 13, pp. 191-215.

ROSS, O.A., CURRAN, M.D., REA, I.M., HYLAND, P., DUGGAN, O., BARNETT, C.R., ANNETT, K., PATTERSON, W., BARNETT, Y.A. and MIDDLETON, D., 2003. HLA haplotypes and TNF polymorphism do not associated with longevity in the Irish. Mechanisms of Ageing and Development, 124, pp. 563-567.

ROSS, O.A., HYLAND, P., CURRAN, M.D., MCILHATTON, B.P., WIKBY, A., JOHANSSON, B., TOMPA, A., PAWELEC, G., BARNETT, C.R., MIDDLETON, D. and BARNETT, Y.A., 2002. Mitochondrial DNA damage in lymphocytes: a role in immunosenescence? Experimental Gerontology (37), pp. 329-340.

HYLAND, P., BARNETT, C.R., PAWELEC, G. and BARNETT, Y.A., 2001. Age-related accumulation of oxidative DNA damage and alterations in levels of p16INK4a/CDKN2a, p21WAF1/CIP1/SD11 and p27KIP1 in human CD4+ T cell clones in vitro. Mechanisms of Ageing and Development, 122, pp. 1151-1167.

HYLAND, P., DUGGAN, O., TURBITT, J., COULTER, J., WIKBY, A., JOHANSSON, B., TOMPA, A., BARNETT, C. and BARNETT, Y., 2001. Nonagenarians from the Swedish NONA Study have increased plasma antioxidant capacity and similar levels of DNA damage in peripheral blood mononuclear cells compared to younger control subjects. Experimental Gerontology (37), pp. 465-473.

HYLAND, P., HIPKISS, A., BARNETT, C.R. and BARNETT, Y.A., 2000. The effects of carnosine on levels and types of DNA damage and lifespan. Mechanisms of Ageing and Development, 121, pp. 203-215.

HYLAND, P., MCKINNEY, M., CURRAN, M., MIDDLETON, D., MCKENNA, P.G. and BARNETT, Y.A., 2000. An investigation of the effect of thymidine kinase deficiency on the spontaneous mutational spectrum at the APRT gene locus in Friend erythroleukaemia cells. Environmental and Molecular Mutagenesis, 36, pp. 87-96.

BARNETT, Y., KING, C., BRISTOW-CRAIG, H., WARNOCK, C., HYLAND, P., GILLESPIE, E., REA, M., MIDDLETON, D., CURRAN, M., PAWELEC, G. and BARNETT, C., 1999. Age-related increases in DNA damage and mutations in T cells in vivo and in vitro: contributors to alterations in T cell-mediated immune responses. In: G. PAWELEC, ed., EUCAMBIS: immunology and ageing in Europe. IOS Press, pp. 54-66. ISBN 9051994346

HYLAND, P., MCKINNEY, M.W., KEEGAN, A.L., MCKENNA, P.G., CURRAN, M.D., MIDDLETON, D. and BARNETT, Y.A., 1997. Sequence analysis of spontaneously-arising mutations at the APRT locus in wild-type and thymidine kinase-deficient Friend cells: evidence of strand slippage misalignment mechanism in formation of deletions. Biochemical Society Transactions, 25 (1), 127S.